Abstract
| Original language | English |
|---|---|
| Pages (from-to) | 1103-1112 |
| Number of pages | 10 |
| Journal | Journal of Sports Medicine and Physical Fitness |
| Volume | 56 |
| Issue number | 10 |
| Early online date | 11 Sep 2015 |
| Publication status | Published - Oct 2016 |
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Influence of recovery intensity on oxygen demand and repeated sprint performance. / Yamagishi, T.; Babraj, John A.
In: Journal of Sports Medicine and Physical Fitness, Vol. 56, No. 10, 10.2016, p. 1103-1112.Research output: Contribution to journal › Article
TY - JOUR
T1 - Influence of recovery intensity on oxygen demand and repeated sprint performance
AU - Yamagishi, T.
AU - Babraj, John A.
PY - 2016/10
Y1 - 2016/10
N2 - AIM: This study aimed to determine effects of recovery intensity (passive, 20, 30 and 40%V̇ O2peak) on oxygen uptake kinetics, performance and blood lactate accumulation during repeated sprints. METHODS: 7 moderately-trained male participants (V̇O2peak: 48.1 ± 5.1 ml·kg-1·min-1) performed 4 x 30-s repeated Wingate tests on 4 separate occasions. RESULTS: Recovery of V̇ O2 between sprints was prolonged with recovery intensity (time required to reach 50% V̇O2peak: Passive: 50 ± 9; 20%: 81 ± 17; 30%: 130 ± 43; 40%: 188 ± 62 sec, P<0.001), while V̇O2-to-sprint work ratio was mainly increased by the higher intensities (Passive: 138 ± 17; 20%: 149 ± 14; 30%: 159 ± 15; 40%: 158 ± 17 ml·min-1·kJ-1, P=0.001). The decline in peak power tended to be greater in the higher intensity conditions during sprint 2 (Passive: 7.4 ± 5.4; 20%: 5.8 ± 7.9; 30%: 12.7 ± 7.4; 40%: 12.7 ± 5.5%, P=0.052), whereas average power was less decreased with recovery intensity during sprint 4 (Passive: 22.4 ± 8.9; 20%: 19.9 ± 6.1; 30%: 18.4 ± 7.3; 40%: 16.6 ± 6.2%, P=0.036). Blood lactate was not different with recovery intensity (P=0.251). CONCLUSION: The present study demonstrated that while the higher recovery intensities induce prolonged oxygen recovery and impaired peak power restoration during the initial sprints, those intensities provide a greater aerobic contribution to sprint performance, resulting in better power maintenance during the latter sprints.
AB - AIM: This study aimed to determine effects of recovery intensity (passive, 20, 30 and 40%V̇ O2peak) on oxygen uptake kinetics, performance and blood lactate accumulation during repeated sprints. METHODS: 7 moderately-trained male participants (V̇O2peak: 48.1 ± 5.1 ml·kg-1·min-1) performed 4 x 30-s repeated Wingate tests on 4 separate occasions. RESULTS: Recovery of V̇ O2 between sprints was prolonged with recovery intensity (time required to reach 50% V̇O2peak: Passive: 50 ± 9; 20%: 81 ± 17; 30%: 130 ± 43; 40%: 188 ± 62 sec, P<0.001), while V̇O2-to-sprint work ratio was mainly increased by the higher intensities (Passive: 138 ± 17; 20%: 149 ± 14; 30%: 159 ± 15; 40%: 158 ± 17 ml·min-1·kJ-1, P=0.001). The decline in peak power tended to be greater in the higher intensity conditions during sprint 2 (Passive: 7.4 ± 5.4; 20%: 5.8 ± 7.9; 30%: 12.7 ± 7.4; 40%: 12.7 ± 5.5%, P=0.052), whereas average power was less decreased with recovery intensity during sprint 4 (Passive: 22.4 ± 8.9; 20%: 19.9 ± 6.1; 30%: 18.4 ± 7.3; 40%: 16.6 ± 6.2%, P=0.036). Blood lactate was not different with recovery intensity (P=0.251). CONCLUSION: The present study demonstrated that while the higher recovery intensities induce prolonged oxygen recovery and impaired peak power restoration during the initial sprints, those intensities provide a greater aerobic contribution to sprint performance, resulting in better power maintenance during the latter sprints.
M3 - Article
VL - 56
SP - 1103
EP - 1112
JO - Journal of Sports Medicine and Physical Fitness
JF - Journal of Sports Medicine and Physical Fitness
SN - 1827-1928
IS - 10
ER -