Vitamin E supplementation does not alter physiological performance at fixed blood lactate concentrations in trained runners

James N. Cobley, K. Marrin

Research output: Contribution to journalArticle

4 Citations (Scopus)

Abstract

The aim of the study was to determine the direction of change in performance variables at fixed blood lactate concentrations following vitamin E (VE) supplementation. In a paired-matched design twelve (male: N.=8; female: N.=4) trained runners were allocated to a VE (N.=6; 268 mg·d-1) or placebo (N.=6; glucose: 30 mg·d-1) group for 35 days. Participants completed a discontinuous incremental exercise test, pre and post supplementation, to determine peak oxygen uptake (VO2peak) running velocity and percentage of peak oxygen uptake (%(VO2peak) at the lactate threshold (TLAC) and the onset of blood lactate accumulation (OBLA). Participants maintained a standardised training regime throughout the supplementation period. VE supplementation failed to significantly enhance velocity at TLAC (P=0.91) and OBLA (P=0.22) compared to a placebo treatment. Analogously, VE did not significantly enhance %(VO2peak) at TLAC (P=0.85) and OBLA (P=0.71) compared to a placebo treatment. Whilst VE supplementation did not enhance performance it did not impair performance compared to a placebo. Training significantly enhanced velocity at TLAC (P=0.00) and OBLA (P=0.05). No training-induced improvements in %VO2peak at TLAC (P=0.06) and OBLA (P=0.40) were observed. Daily VE supplementation for 35 days does not enhance or impair physiological performance at fixed blood lactate concentrations. Long-term VE supplementation for the purposes of performance enhancement is not recommended.
Original languageEnglish
Pages (from-to)63-70
Number of pages8
JournalThe Journal of Sports Medicine and Physical Fitness
Volume52
Issue number1
Publication statusPublished - Feb 2012

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Vitamin E
Lactic Acid
Placebos
Oxygen
Exercise Test
Glucose

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title = "Vitamin E supplementation does not alter physiological performance at fixed blood lactate concentrations in trained runners",
abstract = "The aim of the study was to determine the direction of change in performance variables at fixed blood lactate concentrations following vitamin E (VE) supplementation. In a paired-matched design twelve (male: N.=8; female: N.=4) trained runners were allocated to a VE (N.=6; 268 mg·d-1) or placebo (N.=6; glucose: 30 mg·d-1) group for 35 days. Participants completed a discontinuous incremental exercise test, pre and post supplementation, to determine peak oxygen uptake (VO2peak) running velocity and percentage of peak oxygen uptake ({\%}(VO2peak) at the lactate threshold (TLAC) and the onset of blood lactate accumulation (OBLA). Participants maintained a standardised training regime throughout the supplementation period. VE supplementation failed to significantly enhance velocity at TLAC (P=0.91) and OBLA (P=0.22) compared to a placebo treatment. Analogously, VE did not significantly enhance {\%}(VO2peak) at TLAC (P=0.85) and OBLA (P=0.71) compared to a placebo treatment. Whilst VE supplementation did not enhance performance it did not impair performance compared to a placebo. Training significantly enhanced velocity at TLAC (P=0.00) and OBLA (P=0.05). No training-induced improvements in {\%}VO2peak at TLAC (P=0.06) and OBLA (P=0.40) were observed. Daily VE supplementation for 35 days does not enhance or impair physiological performance at fixed blood lactate concentrations. Long-term VE supplementation for the purposes of performance enhancement is not recommended.",
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Vitamin E supplementation does not alter physiological performance at fixed blood lactate concentrations in trained runners. / Cobley, James N.; Marrin, K.

In: The Journal of Sports Medicine and Physical Fitness, Vol. 52, No. 1, 02.2012, p. 63-70.

Research output: Contribution to journalArticle

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AB - The aim of the study was to determine the direction of change in performance variables at fixed blood lactate concentrations following vitamin E (VE) supplementation. In a paired-matched design twelve (male: N.=8; female: N.=4) trained runners were allocated to a VE (N.=6; 268 mg·d-1) or placebo (N.=6; glucose: 30 mg·d-1) group for 35 days. Participants completed a discontinuous incremental exercise test, pre and post supplementation, to determine peak oxygen uptake (VO2peak) running velocity and percentage of peak oxygen uptake (%(VO2peak) at the lactate threshold (TLAC) and the onset of blood lactate accumulation (OBLA). Participants maintained a standardised training regime throughout the supplementation period. VE supplementation failed to significantly enhance velocity at TLAC (P=0.91) and OBLA (P=0.22) compared to a placebo treatment. Analogously, VE did not significantly enhance %(VO2peak) at TLAC (P=0.85) and OBLA (P=0.71) compared to a placebo treatment. Whilst VE supplementation did not enhance performance it did not impair performance compared to a placebo. Training significantly enhanced velocity at TLAC (P=0.00) and OBLA (P=0.05). No training-induced improvements in %VO2peak at TLAC (P=0.06) and OBLA (P=0.40) were observed. Daily VE supplementation for 35 days does not enhance or impair physiological performance at fixed blood lactate concentrations. Long-term VE supplementation for the purposes of performance enhancement is not recommended.

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